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Bo PengPh.D.

Associate Professor

Research areas: Neuroscience, neuroimmunology

Email:bo.peng1@siat.ac.cn

Dr. Bo Peng obtained his PhD degree from The University of Hong Kong. He mainly focused on the origin and function of microglia in the central nervous system, including the brain and retina. So far, he has published peer-reviewed papers in Nature Neuroscience (corresponding author), Cell Discovery (corresponding author) and Journal of Neuroscience (first author). Besides, he is the reviewer of 4 journals. In 2015, he has joined Shenzhen Institutes of Advanced Technology at Chinese Academy of Sciences as a PI and associate professor. He is also the associate director of Center for Micro Nano Systems and Bionic Medicine.

1. The origin of repopulated microglia in the brain, Shenzhen Science and Technology Research Program, PI, 500,000 CNY, 20180401-20200331, JCYJ20170818163320865.

2. The origin and function of repopulated microglia in the central nervous system, The director foundation of Shenzhen Institutes of Advanced Technology, Chinese Academy of Sciences, PI, 1,000,000 CNY, 20180501-20200531, Y8G007.

3. The origin of repopulated microglia in the adult mouse retina, National Natural Science Foundation of China, PI, 200,000 CNY, 20170101-20191231, 31600839.

4. The strategy of electrical stimulation for retinal prosthesis, National Key R&D Program of China, PI, 1,000,000 CNY, 20180501-20180531, Y8G007.

5. The role of microglial polarization and repopulation in glaucoma, Shenzhen Science and Technology Research Program, PI, 400,000 CNY, 20170525-20190531, JCYJ20170307171222692. 

6. Director funding, Shenzhen Institutes of Advanced Technology, Chinese Academy of Sciences, PI, 1,000,000 CNY, 20180501-20180531, Y8G007.


1. Huang Y. #, Xu Z. #, Xiong S., Sun F., Qin G., Hu G., Wang J., Zhao L., Liang Y.X., Wu T, Lu Z, Humayun M.S., So K.F., Pan Y., Li N., Yuan T.F.*, Rao Y.* and Peng B.* (2018) Repopulated microglia are solely derived from the proliferation of residual microglia after acute depletion. Nature Neuroscience 21, 520-540.

(# co-first authors) (* corresponding authors)

(Featured article. Highlighted by News & Views in the same issue of Nature Neuroscience, Rossi, F., and Lewis, C. (2018). Microglia's heretical self-renewal. Nature Neuroscience 21, 455-456. Also highlighted by the cover of the same issue of Nature Neuroscience)


2. Huang Y. #, Xu Z. #, Xiong S., Qin G., Sun F., Yang J, Yuan T.F. Zhao L, Wang K, Liang Y.X., Fu L., Wu T, Lu Z, So K.F., Rao Y.* and Peng B.* (2018) Dual origins of retinal microglia in the model of microglia repopulation. Cell Discovery 4, 9.

(# co-first authors) (* corresponding authors)


3. Peng B., Xiao J., Wang K., So K.F., Tipoe G.L., and Lin B. (2014). Suppression of microglial activation is neuroprotective in a mouse model of human retinitis pigmentosa. J Neurosci 34, 8139-8150. 


4. Yuan T.F.,Peng B.*, MachadoS., and Arias-Carrion O. (2015). Morphological bases of neuronal hyperexcitability in neurodegeneration. CNS Neurosci Ther 21, 867-869. (* corresponding author)


5. Wang K., Peng B., and Lin B. (2014). Fractalkine receptor regulates microglial neurotoxicity in an experimental mouse glaucoma model. Glia 62, 1943-1954.


6. Liu C., Mao K., Zhang M., Sun Z., Hong W., Li C., Peng B., and Chang Z. (2008). The SH3-like domain switches its interaction partners to modulate the repression activity of mycobacterial iron-dependent transcription regulator in response to metal ion fluctuations. J Biol Chem 283, 2439-2453.


Lab Website: http://www.bopenglab.com

Google Scholar: https://scholar.google.com/citations?user=s4jcKMgAAAAJ&hl=en

ORCID: https://orcid.org/0000-0003-4183-5939


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